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I have essential thrombocytemia and have been on Hydrea for more than 5 years. The dosage differs from time to time, according to my blood tests. Actually I undergo blood tests every months since the time I was diagnosed with the disease. This is not the first thrombocytemia medication I am on, but this is by far the best one. With Hydrea I have least side effects and it provides excellent results on my platelets level. However I have some nausea and occasional headaches I feel much better with Hydrea than with any other med. I do hope that one day I will be able to find a more effective treatment that will help me conquer the illness but in general I am happy with Hydrea as it makes my quality of life so much higher. My doctor is also satisfied with the results we achieve when on Hydrea and he plans to continue this treatment course. I wish him lots of luck!)




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Pseudo sign of Leser Trelat: Increase in size of seborrheic keratoses as seen in sign of Leser Trelat can also be seen after use of cytarabine for the treatment of acute myelogenous leukemia.

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Every body should know his or her genotype especially from primary school children to secondary schools; it would go a long way to reduce the spread of sickle cell disorder in Nigeria, Africa and world at large.

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NIH-supported research projects at the Center focus on the genetic modulation of sickle cell disease, fetal hemoglobin (HbF) regulation in sickle cell disease, the biology and treatment of pulmonary hypertension related to sickle cell disease, and the development of new agents that modulate HbF expression in sickle cell disease and thalassemia.

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Angioid streaks resemble blood vessels, thus their name, and typically emanate radially from the optic nerve.

In this splicing reaction, the ribozyme recognized the sickle beta-globin transcript by basepairing to an accessible region of the RNA upstream of the mutant nucleotide via an internal guide sequence (IGS), cleaved the sickle beta-globin RNA, released the cleavage product containing the mutation, and spliced on the revised sequence for the globin transcript.

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Three of these patients were subsequently treated with daily doses of oral hydroxyurea.

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Deletions of variable extent of the beta gene or of the HBB cluster that result in beta-thalassemia or in the complex beta-thalassemias, called gammadeltabeta-thalassemia and deltabeta-thalassemia, are rare causes of beta-thalassemia and testing that deletions is available clinically by using MPLA.

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Although hematologic changes indicative of the disorder are evident as early as the age of 10 weeks, clinical characteristics of SCD generally do not appear until the second half of the first year of life, when fetal Hb levels decline sufficiently for abnormalities caused by HbS to manifest.

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By initiating hydroxyurea prior to complete engraftment with sickle hematopoiesis and the development of severe organ damage, the potential to observe effects from drug treatment were optimized.

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Differentiation of thalassemia major from thalassemia intermedia at presentation is a difficult and critical issue that should be strongly pursued, because it may avoid unnecessary transfusions in thalassemia intermedia and start early transfusions in thalassemia major.

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This means that there it is less likely for there to be end-organ damage related to the eosinophilia itself.

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After being administered, hydroxyurea blocks the ribonucleotide reductase system where it interferes with the conversion of ribonucleotides into deoxyribonucleotides.

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Treatment options for hydroxyurea-refractory disease complications in myeloproliferative neoplasms: JAK2 inhibitors, radiotherapy, splenectomy and transjugular intrahepatic portosystemic shunt.

Quantitative analysis of erythrocytes containing fetal hemoglobin (F cells) in children with sickle cell disease.

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We assessed penile rigidity during sleep and the relationship of sleep abnormalities with priapism in adults with sickle cell disease.

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This report is based on events that occurred between the start of treatment on January 28, 1992, and June 30, 1994, 10 months before the planned end of the study.

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For induced HbF levels, genetic approaches using candidate single-nucleotide polymorphisms (SNPs) have identified some of these same loci as being also associated with induction.

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SCD is (especially the atypical serotypes Salmonella typhimurium, Salmonella enteritidis, Salmonella choleraesuis and Salmonella paratyphi B), followed by and Gram-negative enteric bacilli perhaps because intravascular sickling of the bowel leads to patchy ischaemic infarction.

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On the other hand, in case of an affected fetus and a previous normal child, the couple may decide to continue the pregnancy and pursue BMT later, using the normal child as the donor.

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Effects on baseline HbF from a SNP in each these two genes were additive and were associated with two-fold higher HbF for patients with favorable alleles in both loci.

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Whereas mutations, genetic drift, and natural selection take place over many generations, epidemics can spread through large populations within a few days or weeks.

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